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PMID 4720899. The monohydroxytryptamines, 6-HT and 7-HT when administered intraventricularly to rats in doses of 100 or 200 μg of free base, precipitated only a brief lowering of mind 5-HT and NA (BAUMGARTEN et al., in preparation) In distinction, 5,7-DHT, 4,5-DHT and 4-hydroxy-5-methoxytryptamine produced a long-lasting depletion of brain and spinal cord 5-HT and NA (BAUMGARTEN et al., in preparation). The descending order of potency for this impact was 5,7-DHT 4,5-DHT and 4-hydroxy-5-methoxytryptamine, the compounds being administered through the lateral ventricle in doses of fifty or a hundred μg of free base. All three compounds exerted a a lot greater impact on CNS 5-HT than on NA levels. The differential potency of 5,7-DHT, 4,5-DHT and 4-hydroxy-5-methoxytryptamine in depleting endogenous 5-HT was additionally mirrored in morphological research of the extent of harm precipitated to central 5-HT neurons. The extreme disintegration of terminals containing 5-HT, in contrast to those having NA, and the lesioning of non-terminal axons following 5,7-DHT has been reported earlier (BAUMGARTEN and LACHENSAYER, 1972b) and was shown to be much like that induced by 5,6-DHT (BAUMGARTEN and LACHENMAYER, 1972a). The above order of potency of 5,7-DHT, 4,5-DHT and 4-hydroxy-5-methoxytryptamine in causing toxic harm to CNS serotonin neurons doesn`t strictly correspond to their relative affinity for the 5-HT uptake site but quite reflects the chemical lability of the medicine.



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